A text-book of dental histology and embryology, including laboratory directions . n becomesgreatly exaggerated, and changes which would ordinarilyproduce no effect will produce acute hyperemia. When theirritability of the fibrils and the odontoblasts has been greatlyincreased by the action of irritating agents, as in the progressof caries, or when the thickness of the protecting dentinehas been greatly reduced, as in abrasion, or when consider-able masses of gold are separated from the pulp only by athin layer of dentine, the same conditions result. In this stage of hyperemia the only change i
A text-book of dental histology and embryology, including laboratory directions . n becomesgreatly exaggerated, and changes which would ordinarilyproduce no effect will produce acute hyperemia. When theirritability of the fibrils and the odontoblasts has been greatlyincreased by the action of irritating agents, as in the progressof caries, or when the thickness of the protecting dentinehas been greatly reduced, as in abrasion, or when consider-able masses of gold are separated from the pulp only by athin layer of dentine, the same conditions result. In this stage of hyperemia the only change in the tissuethat can be observed under the microscope is the distentionof the capillaries and veins, and as soon as the pain haspassed the tissue returns to a normal condition. It is apparently, therefore, a functional disturbance, due ACUTE HYPEREMIA 221 to the increased irritability of the cytoplasm of the fibrils,odontoblasts, and probably also of the nerve endings. Therational treatment for such conditions is the removal of the Fig. 164 >*> •: fj *SM f ? | )** ^l. Acute hyperemia, higher power. 222 STRUCTURAL CHANGES IN PATHOLOGY OF PULP irritation which has caused the irritability, and the completeprotection of the tooth from thermal change, until the restrestores the normal function. In order to observe the structural changes, the tooth mustbe extracted during the paroxysm of pain, and should becracked and dropped at once into a fixing fluid, allowed toremain there for about twenty-four hours, when the pulpcan be removed from the pulp chamber and embedded andsectioned. In this way the injection of the bloodvessels willbe preserved, and all of the capillaries and veins will befound crowded with corpuscles, and their distention will beproportionate to the severity of pain at the time of theextraction. Acute hyperemia has two possible terminations asidefrom recovery: (1) If often repeated, it may pass over intochronic hyperemia; (2) if severe enough, it may end ininf
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