. Fig. 157.—Drawing from a microscopic preparation, showing the glandular hyper- plasia of the mucosa of a gall-duct caused by the common liver fluke {Fasciola hepatica). a, Hypertrophied submucosa ; b, interstitial connective tissue; c, com- pressed lobule ; d, lumen of the gall-duct—thickened fibrous wall of the gall-duct. (After Schaper, 1890, PI. I., Fig. 2.) fact probably explains to some degree the wide geographical distribution of the parasite. Symptoms. The symptomatology of this disease may clinically be divided into three well-marked periods: I. Primary period. The primary phase comm
. Fig. 157.—Drawing from a microscopic preparation, showing the glandular hyper- plasia of the mucosa of a gall-duct caused by the common liver fluke {Fasciola hepatica). a, Hypertrophied submucosa ; b, interstitial connective tissue; c, com- pressed lobule ; d, lumen of the gall-duct—thickened fibrous wall of the gall-duct. (After Schaper, 1890, PI. I., Fig. 2.) fact probably explains to some degree the wide geographical distribution of the parasite. Symptoms. The symptomatology of this disease may clinically be divided into three well-marked periods: I. Primary period. The primary phase commences with the pene- tration of the embryos of the parasite into the body, firstly into the intestine, and then into the liver by ascending the bile ducts. This phase occurs during the last months of the year, October, November, and December, and is rarely accompanied by alarming symptoms. At this time the sheep appear in good health, the summer being over, and the
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Keywords: ., bookcentury1900, bookdecade1920, booksubjectveterin, bookyear1920
