Archives of internal medicine . Fig. 14.—Adenoma of intermediate type but closely ajjpioximating the truefetal adenoma. Ndtc tlie capsule with the tumor tissiie on one side and theoriginal thyroid (in tlic otlicr Fig. 15.—True fetal adenoma. Section taken from central part of tumor wherefollicles are more separated. Note size, shape and arrangement of follicles, thelining epithelium and the absence of colloid. DAVID MAIUXE—C. //. LEX RANT 517 a general increase in its blood-supply, a general decrease in the stainablecolloid and a general change from cuboidal to columnar epithelium. Ifth
Archives of internal medicine . Fig. 14.—Adenoma of intermediate type but closely ajjpioximating the truefetal adenoma. Ndtc tlie capsule with the tumor tissiie on one side and theoriginal thyroid (in tlic otlicr Fig. 15.—True fetal adenoma. Section taken from central part of tumor wherefollicles are more separated. Note size, shape and arrangement of follicles, thelining epithelium and the absence of colloid. DAVID MAIUXE—C. //. LEX RANT 517 a general increase in its blood-supply, a general decrease in the stainablecolloid and a general change from cuboidal to columnar epithelium. Ifthese changes continue to the stage of cell proliferation, the conditionis properly designated as hyperplasia. There are all degrees of the abnor-mal cell growth from the slightest departure from normal (hypertrophy)to the marked proliferation (hyperplasia). Hyperplasia is the anatomical evidence of increased physiologicalactivity of the gland, and the instances in which visible goiters appear areonly accidents, so to speak, since the great majority of all hyperplasias inall animals never reach that degree of proliferation which could be clin-ically diagnosed as goiter. Clinically, therefore, goiter is relativ
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