Studies in cardiac pathology . en pointed out by Samways,who in 36 severe cases of mitral stenosis found hypertrophy in 26,whereas dilatation occurred in only 14, in 11 of which it was asso-ciated with hypertrophy. This process of dilatation manifestsitself first in the auricular appendix, owing to the relativelyunyielding structures by which the auricle proper is surrounded(the aorta, vertebrae, and esophagus behind, the other heart cham-bers in front, the diaphragm and liver below, and the aortic archand pulmonary artery above). (See Figs. 45, 46, and 47.) Thrombosis, either ante mortem or p
Studies in cardiac pathology . en pointed out by Samways,who in 36 severe cases of mitral stenosis found hypertrophy in 26,whereas dilatation occurred in only 14, in 11 of which it was asso-ciated with hypertrophy. This process of dilatation manifestsitself first in the auricular appendix, owing to the relativelyunyielding structures by which the auricle proper is surrounded(the aorta, vertebrae, and esophagus behind, the other heart cham-bers in front, the diaphragm and liver below, and the aortic archand pulmonary artery above). (See Figs. 45, 46, and 47.) Thrombosis, either ante mortem or post mortem, of either ofthese chambers occurs frequently, owing to circulatory stagnationand roughening of the endocardium bj^ vegetations. (See Fig. 9.)Such a state of affairs is a prolific source of emboli, and adds muchto the weakness of the heart which is manifest during intercurrentinfections, such as influenza, and which is often attributed to Hiischfelder and Wolfsohn: Johns Hopkhis Hosp. Bulletin, xix, 190S. :;- — to.
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