. Dental materia medica and therapeutics; with special reference to the rational application of remedial measures to dental diseases ... kaler Infectionen mittelst Hyperamie, Wurzburger Abhandlungen,Vol. VI, No. 6. 408 PHYSICAL THERAPEUTICS. that the normal osmotic pressure of the tissue fluids amounts toabout to atmospheres, which, when expressed relative tothe freezing point of a physiologic salt solution, equals ° ° C. Under normal conditions the osmotic pressure ispromptly regulated by the organism; probably, according to Mas-sart, through specific nerves—that is, the nor
. Dental materia medica and therapeutics; with special reference to the rational application of remedial measures to dental diseases ... kaler Infectionen mittelst Hyperamie, Wurzburger Abhandlungen,Vol. VI, No. 6. 408 PHYSICAL THERAPEUTICS. that the normal osmotic pressure of the tissue fluids amounts toabout to atmospheres, which, when expressed relative tothe freezing point of a physiologic salt solution, equals ° ° C. Under normal conditions the osmotic pressure ispromptly regulated by the organism; probably, according to Mas-sart, through specific nerves—that is, the normal equilibrium ofthe isotonic index of the blood and tissue fluids remain station-ary. In pathologically altered tissues the composition is continu-ally interfered with, and usually results in a marked increase ofthe osmotic pressure—hyperisotonicity. Increased osmotic pres-sure produces pronounced morphologic changes in the cells, andis largely responsible for the resultant pain, followed by inflam-mation, within the affected area. According to Bitter1 the va-rious changes in tissues, if a simple abscess is taken as an ex-. FlGURE 51. Schematic Drawing1 of an Abscess. The abscess and the surrounding infiltrated area showthe various degrees of osmotic pressure, a, abscess; b, hyperemic zone; c, manifest edema;d, latent edema. ample, may be described as follows: In the center of the puscavity the osmotic pressure may reach a density of ° to ° C.(° being normal), but in the surrounding hyperemic zonethe pressure is less, gradually diminishing in the manifest edema,and becoming less and less toward the periphery until normal pres-sure is reached. Aside from these quantitative changes within theinflamed area, qualitative changes of the constituents of the exu-dates undoubtedly have some important significance. The natureof these latter changes is at present too obscure to allow any defi-nite statements to be made. Whenever living tissue is injured—whether by m
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