. Transactions . e are no blood-corpuscles within the lunK>n. vein is enormously thickened, not by endothelial pro-liferation, but by increase in the breadth of the connective-tissue wall (Fig. 48). Farther back collaterals may be seenentering the Iuukmi of the vein from the trabecula) of thenerve, so that tlu^ lunuMi Liradually becomes larger whilethe wall remains very thick. Finally (but this doesnot occur for a considerable distance), the vein regainsits normal size and ceases to have unduly thick after this it begins to leave the artery, and soon 168 DISEASES OF THE RETINA
. Transactions . e are no blood-corpuscles within the lunK>n. vein is enormously thickened, not by endothelial pro-liferation, but by increase in the breadth of the connective-tissue wall (Fig. 48). Farther back collaterals may be seenentering the Iuukmi of the vein from the trabecula) of thenerve, so that tlu^ lunuMi Liradually becomes larger whilethe wall remains very thick. Finally (but this doesnot occur for a considerable distance), the vein regainsits normal size and ceases to have unduly thick after this it begins to leave the artery, and soon 168 DISEASES OF THE RETINA AND CHOROID. reaches the periphery of the nerve ; a small branch con-tinues to accompany the artery. The vein is here quitenormal. It is not clear that at the obliterated portion ofthe vein the collaterals among the trabeculae of the nerveare enlarged; they are, however, enlarged farther back.^.t^The central artery shows for the most part little changebeyond a slight endarteritis. At the place, however, Fin.
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Keywords: ., bookcentury1800, bookdecade1880, bookpu, booksubjectophthalmology
