American journal of physiology . Fig. I. Curves of the tension in the aorta (upper tracing) and the quickness of flow in theright coronary artery of the horse, simultaneously recorded (Rebatels Fig. 3, page 25). into the coronary artery, or to a sudden diminution of the peripheralresistance in the intramural vessels (p. 27), To determine the originof the second current, the tension and the quickness of flow in thecoronary artery were recorded simultaneously. It was then seen thatthe tension curve was like that of every other artery, and presented nosecondary rise or other feature that could ac
American journal of physiology . Fig. I. Curves of the tension in the aorta (upper tracing) and the quickness of flow in theright coronary artery of the horse, simultaneously recorded (Rebatels Fig. 3, page 25). into the coronary artery, or to a sudden diminution of the peripheralresistance in the intramural vessels (p. 27), To determine the originof the second current, the tension and the quickness of flow in thecoronary artery were recorded simultaneously. It was then seen thatthe tension curve was like that of every other artery, and presented nosecondary rise or other feature that could account for the secondaryaugmentation in the quickness of flow. Thus led to a variation in theperipheral resistance, Rebatel concluded that the primary blood-wave. Fir,. 2. Curves of the tension and quickness of flow in the right coronary artery of thehorse, simultaneously recorded (Rebatels Fig. 8, page 29). penetrates with difiiculty into the intramural branches during systole,because of their compression by the contracting cardiac muscle, butwhen the relaxation of the ventricle opens the peripheral vessels, thepent stream rushes suddenly forwards, and thus produces the second,or diastolic, rise in the curve of the haemodromograph. 150 W. T. Porter. Rebatel himself does not accept this hypothesis unreservedly. Hisattitude is that of M. Marey,^ who, on being shown the curves, admittedthat the first proposition, namely, the filling of the coronary arteriesin systole, is incontestable, while the second, namely, that the increasein quickness of flow is due to the qpening of the intramural vessels bythe relaxation of the ventricle is only very probable and legitimatelydeduced. An analysis of Rebatels tracings (Fig. 2) suggests thateven this qualifi
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