. Internal medicine; a work for the practicing physician on diagnosis and treatment, with a complete Desk index. lestjomt in locomotor —Lloyd. kncc-jcrks, but without impairment of gait. This form may persist for many years, finally developing reported a case which preserved this type for fifteen years, andGowers mentions one in which optic atrophy had existed for twenty yearsbefore the onset of incoordination. This type is peculiarly liable to arthrop-athies. Locomotor ataxia sometimes coexists with general paresis. It mayprecede that disease, as is the more common way,
. Internal medicine; a work for the practicing physician on diagnosis and treatment, with a complete Desk index. lestjomt in locomotor —Lloyd. kncc-jcrks, but without impairment of gait. This form may persist for many years, finally developing reported a case which preserved this type for fifteen years, andGowers mentions one in which optic atrophy had existed for twenty yearsbefore the onset of incoordination. This type is peculiarly liable to arthrop-athies. Locomotor ataxia sometimes coexists with general paresis. It mayprecede that disease, as is the more common way, or in some cases it mayfollow it. There is a juvenile tabes, which is the result of hereditary disease, however, is usually one of adult life, the initial symptoms gen-erally showing themselves between the ages of 30 and 40 years. It is notlimited to any race or country, but is seen wherever syphilis abounds; andthe statement that locomotor ataxia does not occur in the negro race, iserroneous. It is not so common among women in any race as among has been seen in both husband and LOCOMOTOR ATAXIA. 701 Diagnosis.—Jjocoiiiotoi- alaxia is to he dist inmiislird Irom multiploneuritis by the ful^urant pains, the crises, the pupilhiry changes, the bladdersymptoms, and tlie absence of true paralysis with atrophy and the; reactionsof degeneration. There is a pseKclotalxs due to nndtiplc neuritis, in whichataxia is marked, but the history of the case, the llaecid paralysis, withatrophy ami electrical chan<res, the painful nerve-trunks and nmscles as wellas the absence of the optic atrophy and pupillary changes, serve to distin-guish it. Muscular atrophy with loss of ])()wer may occur in advancijd stag(;sof tabes, but the other tabetic symptoms and the history of the case shouldprevent error. From syringomyelia, in which there sometimes occurs anataxic type, tabes is distinguished by the absence of the dissociation syn-drome, of the scoliosis, of the spas
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Keywords: ., bookcentury1900, bookdecade1920, booksubjectmedicine, bookyear192
