Diseases of cattle, sheep, goats Diseases of cattle, sheep, goats and swine diseasesofcattl00mous Year: 1905 168 COLIC. or by strangulation of an intestinal loop by fibrous bands resulting from chronic peritonitis, etc. Of these various causes, the three prin- cipal may here be described:— (1.) Tearing of the mesentery. As a result of mechanical violence the epiploon or mesentery becomes fissured, and the peristaltic move- ments cause a loop of intestine to pass through and become fixed in the fissure. If the opening is narrow, as is usually the case, the base of the intestinal loop, riding
Diseases of cattle, sheep, goats Diseases of cattle, sheep, goats and swine diseasesofcattl00mous Year: 1905 168 COLIC. or by strangulation of an intestinal loop by fibrous bands resulting from chronic peritonitis, etc. Of these various causes, the three prin- cipal may here be described:— (1.) Tearing of the mesentery. As a result of mechanical violence the epiploon or mesentery becomes fissured, and the peristaltic move- ments cause a loop of intestine to pass through and become fixed in the fissure. If the opening is narrow, as is usually the case, the base of the intestinal loop, riding on the lower lip of the slit, becomes con- stricted by the margins of the opening through which it has passed. (2.) In pelvic hernia a loop of intestine passes between the spermatic cord and the walls of the pelvis. The fissure in this case is in the serous fold which supports the large testicular arteries and the vas deferens. The fold is often ruptured during castration, especially during the practice of ' bistournage,' in conse- quence of traction exercised on the cord. (3.) Pseudo - ligaments and fibrous bands due to chronic peritonitis.—-In local, subacute or chronic peritonitis false membranes may become organised, form- ing fibrous cords or folds connecting the parieto-visceral or inter-visceral surfaces. If by accident a loop of intestine insinu- ates itself beneath one of these fibrous bands, the passage of digestive material is first impeded and then stopped. The intestine becomes engorged, and symp- toms of strangulation soon follow. The symptoms appear suddenly, and are similar to those of inva- gination. They consist of very acute colic, which disappears after ten to twelve hours. The peristaltic movements drive the semi-digested food, whether liquid or gaseous, towards the lower (strangulated) end, from which it cannot escape. It therefore distends the herniated loop and sets up intestinal engorgement. This constitutes the first stage of strangulation, and is
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