Intravenous injection in wound shock : being the Oliver-Sharpey lectures delivered before the Royal College of Physicians of London in May 1918 . Fig. 43.—. of Bicarbonate after Muscle Injury. Experiment similar to that of Fig. 42. a. Hammered legs. b. 25 of 4*2 per cent, bicarbonate. c. Hammered right leg again as the previous injury had been slight. No bone broken. d. 15 bicarbonate. e. An hour and a half after 5 bicarbonate. o. Attacks of respiratory gasps, followed by very shallow Two hours after injury. u8 INTRAVENOUS INJECTION me that lact
Intravenous injection in wound shock : being the Oliver-Sharpey lectures delivered before the Royal College of Physicians of London in May 1918 . Fig. 43.—. of Bicarbonate after Muscle Injury. Experiment similar to that of Fig. 42. a. Hammered legs. b. 25 of 4*2 per cent, bicarbonate. c. Hammered right leg again as the previous injury had been slight. No bone broken. d. 15 bicarbonate. e. An hour and a half after 5 bicarbonate. o. Attacks of respiratory gasps, followed by very shallow Two hours after injury. u8 INTRAVENOUS INJECTION me that lactic acid is innocuous in wound shock (seeFigs. 41 and 42). The injection of sodium bicar-bonate does not prevent the development of shockafter muscle injury (Figs. 32, 43, and 45); whereasthat of gum-saline does (see Figs. 32, 40, 41, and 42).A further test was made by extracting muscles,which had gone into rigor, with boiling salinesolution. A part of the extract was Fig. 43.— of Bicarbonate after Muscle Injury. Cord cut in upper lumbar region. Haemorrhage. a. Legs hammered. Resp. 80. b. Bled 27 per cent. c. Partial recovery, but respirations rapid, about 82, with gasps at intervals. d. After 50 gum-saline. Resp. 36. e. One hour and ten minutes later. Probably gum insufficient in amount. Respiration rather 15 half-normal Twenty minutes later, 15 Half an hour this experiment gum had only a temporary effect, although it wasbetter than bicarbonate. IN WOUND SHOCK 119 The effects on the blood pressure and on theintestinal vessels produced by equal doses were thesame. Hence the vaso-dilator substance is notlactic acid (Fig. 44). Vincent and Sheen(1903) have describedsimilar vaso-dilator effectsfrom muscle extracts. Ac-cording to Sand (1917),toxic products are formedby the disintegration ofinjured muscles, whetherproduced by the directmechanical effect of shellfragments, or by gas infe
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