. The Biochemical journal. Biochemistry. DEFICIENT NUTRITION AND IMMUNITY 185 of experiments were devoted to this subject. In the first set a low intake of caseinogen was studied, in the second the action of gliadin as the sole source of protein in the diet. Osborne and Mendel [1915] were the first to demonstrate that if caseinogen formed 18 % of a diet it was adequate to ensure normal growth in rats; if, however, the content of that protein in the diet was reduced to 9 % the rate of growth of the animals was greatly impaired. The explanation was advanced that cystine was the limiting factor,
. The Biochemical journal. Biochemistry. DEFICIENT NUTRITION AND IMMUNITY 185 of experiments were devoted to this subject. In the first set a low intake of caseinogen was studied, in the second the action of gliadin as the sole source of protein in the diet. Osborne and Mendel [1915] were the first to demonstrate that if caseinogen formed 18 % of a diet it was adequate to ensure normal growth in rats; if, however, the content of that protein in the diet was reduced to 9 % the rate of growth of the animals was greatly impaired. The explanation was advanced that cystine was the limiting factor, caseinogen having a low cystine content. When it forms as much as 18 % of the diet the amount of cystine supphed through its medium is sufficient to satisfy the requirements of the animals, when, however, it forms only 9 % of the diet the amount of cystine consumed becomes deficient and manifests itself by the restricted growth of the animals. This explanation was confirmed by the fact that the addition of cystine to a low caseinogen diet raised its nutritive value to the same level as that of lactalbumin. 900 800- 400 200. Please note that these images are extracted from scanned page images that may have been digitally enhanced for readability - coloration and appearance of these illustrations may not perfectly resemble the original Biochemical Society (Great Britain); University of Liverpool. Biochemical Dept. London [etc. ] Cambridge University Press
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