Pulmonary consumption, pneumonia, and allied diseases of the lungs : their etiology, pathology and treatment, with a chapter on physical diagnosis . Such conglomera-tions of giant-cell systems as those represented in Fig. 23are formed in this , and the extent to which the lungtissue may be involved by such supernumerary growths issometimes very great. A few tubercle nodules are firstformed in a localized area, and then from their borders off-shoots are projected by a process of invagination into theneighboring air-vesicles. The lung becomes almost solidat such parts, from the immense ma
Pulmonary consumption, pneumonia, and allied diseases of the lungs : their etiology, pathology and treatment, with a chapter on physical diagnosis . Such conglomera-tions of giant-cell systems as those represented in Fig. 23are formed in this , and the extent to which the lungtissue may be involved by such supernumerary growths issometimes very great. A few tubercle nodules are firstformed in a localized area, and then from their borders off-shoots are projected by a process of invagination into theneighboring air-vesicles. The lung becomes almost solidat such parts, from the immense mass of tubercles whichare thus called into existence. In the course of time thetubercle outgrowth comes to fill the air-sac, and their wallsbecome contiguous. In this condition we have two processes of disintegrationgoing on. First, that which begins by filling and packingthe alveoli with catarrhal products, and ends by the-latterundergoing caseation and by the formation of cavities; and,second, the absorption of tubercular virus by the lymphatic vessels, and the production of a PATHOLOGY OF PULMONARY CONSUMPTION. 273. tpr Fig. 23. — Showing tubercle of the lung. a, a, a, a, fourtubercles; d, thickened interstitial tissue uniting two tubercles; c,giant-cells; d, giant-cell reticulum ; e, center of tubercle caseating;/,a giant-cell system which has become converted into a mass of fibroustissue.—fJainiiton, 2 74 DISEASES OF THE LUNGS. crop of tubercles along the course of the latter. The firstis a chronic catarrhal pneumonia, and corresponds with thecharacteristics that distinguish that pathologic conditionlaid down above, as the first form; and the second is a gen-uine pulmonary tuberculosis, and corresponds with the sec-ond form of pathologic characteristics already described. What, then, is the destiny of these tubercles? Do theyundergo caseation like the nodules of catarrhal pneumonia,and thus become sources of further infection, or do theyremain
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