. Internal medicine; a work for the practicing physician on diagnosis and treatment, with a complete Desk index. inence due to hypertrophy of the right ven-tricle. The apex beat is commonly displaced but slightly toward the left,and may be indistinct, the visible impulse being at the lower end of thesternum and extending to theleft costal cartilages. The pul-sation of the conns arteriosusmay often be visible at the ster-nal end of the third and fourthleft interspaces. As compensa-tion fails, the impulse loses itspower, and signs of back pressure in the systemic veins appear, as distentionof th
. Internal medicine; a work for the practicing physician on diagnosis and treatment, with a complete Desk index. inence due to hypertrophy of the right ven-tricle. The apex beat is commonly displaced but slightly toward the left,and may be indistinct, the visible impulse being at the lower end of thesternum and extending to theleft costal cartilages. The pul-sation of the conns arteriosusmay often be visible at the ster-nal end of the third and fourthleft interspaces. As compensa-tion fails, the impulse loses itspower, and signs of back pressure in the systemic veins appear, as distentionof the superficial veins, especially the jugulars, with pulsation due to con-traction of the right ventricle and enlargement of the liver. Upon palpationthere is recognized in at least three-fourths of the cases a very distinctthrill. This sign is usually coarse in character, diastolic or presystolic intime, confined to an area above the apex, usually in the fourth and fifthinterspaces, and circumscribed. It is more intense and slightly moreextended during expiration and runs up to a short, sharp apex beat. This. Fig. —Mitral stenosis; carotid trafing.
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Keywords: ., bookcentury1900, bookdecade1920, booksubjectmedicine, bookyear192
