. Oral pathology and practice. A text-book for the use of students in dental colleges and a hand-book for dental practitioners. he one makes progress from withinoutward: the other just the reverse. The one tends toward resolu-tion : the other is progressively degenerative. An abscess is alwaysthe result of a recent lesion : an ulcer is never connected with a freshwound or infection, but has its inception in some old injury ormorbid structural change. It would be difficult to instance agrosser misuse of technical terms than the calling of an alveolarabscess an ulcerated tooth. Professor Kirk ha
. Oral pathology and practice. A text-book for the use of students in dental colleges and a hand-book for dental practitioners. he one makes progress from withinoutward: the other just the reverse. The one tends toward resolu-tion : the other is progressively degenerative. An abscess is alwaysthe result of a recent lesion : an ulcer is never connected with a freshwound or infection, but has its inception in some old injury ormorbid structural change. It would be difficult to instance agrosser misuse of technical terms than the calling of an alveolarabscess an ulcerated tooth. Professor Kirk has demonstrated that a pericemental abscessmay develop in the parenchyma of the membrane; that is, it may be ALVEOLAR ABSCESS. II9 neither supra- nor infra-, but intra-pericemental. (See Figs. 30and 31.) It is indeed probable that such abscesses are more fre-quent than is usually supposed. Most practitioners of experiencehave at some time in their lives drilled into an abscessed tooth andfound a living pulp, which would demonstrate that the lesion wasnot at the foraminal apex. By the study of these conditions Pro- Fig. Transverse Section across Buccal Roots of Fig. 30, showing the Abscess-cavityto be between the pericemental walls. a, a. Hypercementosis. b. Thickened pericementum covering root. bx. Thickened peri-cementum forming external wall of abscess-cavity, c. Abscess-cavity occupying central por-tion of divided pericemental membrane, d. Section through fistulous outlet of abscess. (Kirk.) fessor Kirk believes he has found a common factor of infection tobe the diplococcus of pneumonia, or the pneumococcus of Fried-lander, with occasionally staphylococcus pyogenes aureus as a con-comitant. 120 ORAL PATHOLOGY AND PRACTICE. But such a condition is not that which has usually been denomi-nated alveolar abscess. The common acceptation of the term is thataffection which is the result of inflammation and death of thepulp, its infection, and the consequent inflammation and infe
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