. Electrolytes in biological systems, incorporating papers presented at a symposium at the Marine Biological Laboratory in Woods Hole, Massachusetts, on September 8, 1954. Electrophysiology; Electrolytes; Electrolytes; Electrophysiology; Physiology, Comparative. GEORGE T. SCOTT AND HUGH R. HAYWOOD 43 the pn of the medium caused by photosynthesis and hence an increase in the activity product (K)(OH) outside the cell. In the above experiments the samples were maintained in freely running sea water so that changes in external pH would be insignificant. Influence of lodoacetate in the Light and th
. Electrolytes in biological systems, incorporating papers presented at a symposium at the Marine Biological Laboratory in Woods Hole, Massachusetts, on September 8, 1954. Electrophysiology; Electrolytes; Electrolytes; Electrophysiology; Physiology, Comparative. GEORGE T. SCOTT AND HUGH R. HAYWOOD 43 the pn of the medium caused by photosynthesis and hence an increase in the activity product (K)(OH) outside the cell. In the above experiments the samples were maintained in freely running sea water so that changes in external pH would be insignificant. Influence of lodoacetate in the Light and the Dark: Ulva. The relationship of carbohydrate metabolism to cation regulation was further explored by use of the glycolytic inhibitor Fig. 3. Influence of m/1. lodoacetate on the potassium content of Ulva lactuca in the light and in the dark. Potassium is expressed in terms of cell water. The presence of the inhibitor in a concentration of m/1. results in a marked loss of potassium from the cells over a period of 24 hours in the dark. Control samples taken at the beginning and end of this period were essentially constant in potassium content (fig. 3). In the presence of light the inhibitor is completely ineffective in causing the loss of potassium. Rather, the potassium content of the experimentals temporarily increases over that of the controls. To evaluate further the influence of light on the prevention of the iodo- acetate effect, the concentration of the inhibitor was raised to '^/^- Again light prevented the loss of potassium. Concomitant with the potassium loss caused by the m/1. iodoacetate in the dark, the cellular sodium increases over that of the controls although the condition of darkness alone is sufficient to cause some sodium increase. Illumina- tion again prevents this action of the inhibitor, and sodium is actually some-. Please note that these images are extracted from scanned page images that may have been digitally
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