Clinical electrocardiography . Fig. 67.—Premature ventricular contractions preceding a paroxysm of ven-tricular tachycardia. Electrocardiogram in Derivation Fig. 68.—Paroxysm of ventricular tachycardia. Rate 180. Electrccardograi in Derivation I. ECTOPIC RHYTHMS AND TACHYCARDIAS 91 The auricles and ventricles contract at the same rate, for eachcomplex is identical to the adjacent complex, and if auricles andventricles were contracting at independent rates, the auricularcomplex would at times be superimposed and destroy the contourof the general curve. Rarely does the rate of the ventricle
Clinical electrocardiography . Fig. 67.—Premature ventricular contractions preceding a paroxysm of ven-tricular tachycardia. Electrocardiogram in Derivation Fig. 68.—Paroxysm of ventricular tachycardia. Rate 180. Electrccardograi in Derivation I. ECTOPIC RHYTHMS AND TACHYCARDIAS 91 The auricles and ventricles contract at the same rate, for eachcomplex is identical to the adjacent complex, and if auricles andventricles were contracting at independent rates, the auricularcomplex would at times be superimposed and destroy the contourof the general curve. Rarely does the rate of the ventriclesexceed that of the auricles, as in Palfreys case. In all probability, any condition increasing ventricular irrita-bility is a potential factor in the production of ventricular tachy-cardia, and until more necropsy material is available conclusionswith regard to types of lesions must remain hypothetic. Thegravity of the condition depends, of course, on the degree of myo-cardial damage and the duration of the paroxysms. The mainte-nance of circulation is dependent on ventricular action and not onauricular action, and obviously this abnormal ventricular r
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