General and dental pathology with special reference to etiology and pathologic anatomy; a treatise for students and practitioners . h are more or less phagocytic, while the re-mainder elongate and form fibrous tissue (fibroblasts). Degenerative changes are always present in varying degrees, con-sisting of cloudy swelling, mucoid or fatty degeneration, coagula-tion necrosis, liquefaction or other necrosis. A typical, acute inflammation is therefore characterized by Hyperemia of the affected area, Exudation of serum from the vessels, Emigration of leucocytes, Diapedesis of red blood cells, Proli
General and dental pathology with special reference to etiology and pathologic anatomy; a treatise for students and practitioners . h are more or less phagocytic, while the re-mainder elongate and form fibrous tissue (fibroblasts). Degenerative changes are always present in varying degrees, con-sisting of cloudy swelling, mucoid or fatty degeneration, coagula-tion necrosis, liquefaction or other necrosis. A typical, acute inflammation is therefore characterized by Hyperemia of the affected area, Exudation of serum from the vessels, Emigration of leucocytes, Diapedesis of red blood cells, Proliferative changes in the fixed tissues, Degenerative changes. (Figs. 20, 21, and 22.) There are ten types of inflammation: 100 GENERAL PATHOL* I. Edematous or Serous.—In this type there is an excessivefluid exudation, with little tendency to fibrin formation, andrelatively few cells, due to intense irritation. - steam or cor-rosives: or to mild subacute or chronic irritation, as in serouseffusions. II. Fibrinous.—The exudate is especially rich in fibrin factors,the fibrin appearing as fibrillar, granular or homogeneous de-. Fig. 20.—Acute inflammation. (Mallory.) m ?* %
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Keywords: ., bookcentury1900, bookdecade1920, booksubjectpathology, bookyear19
