Manual of pathology : including bacteriology, the technic of postmortems, and methods of pathologic research . part, through adja-cent communicating capil-laries,or,slightly, if at all,byvenous regurgitation; fromthe congested capillaries ex-travasation of blood intothe connective tissue occurs,and, in addition to the coag-ulation necrosis in progressin the cellular elements ofthe area, a further fibrinousmatting together resultsfrom the associated hem-orrhagic infiltration. Theresulting change constitutes a hemorrhagic infarct, and the process terminating m its lurmaiion isknown as hcmorrha^i
Manual of pathology : including bacteriology, the technic of postmortems, and methods of pathologic research . part, through adja-cent communicating capil-laries,or,slightly, if at all,byvenous regurgitation; fromthe congested capillaries ex-travasation of blood intothe connective tissue occurs,and, in addition to the coag-ulation necrosis in progressin the cellular elements ofthe area, a further fibrinousmatting together resultsfrom the associated hem-orrhagic infiltration. Theresulting change constitutes a hemorrhagic infarct, and the process terminating m its lurmaiion isknown as hcmorrha^ic injarction. The shape ot the area is not alteredby the occurrence of hemorrhagic infiltration. The swelling, however,is more marked; the color is dark red, at times almost black a-v!the density is greatlv increased by the presence of the coblood. The subsequent changes are the same as those occurnngof anemic infarction. It has been held that an anemic or white infarct is but a lat-of the hemorrhagic form and that it is dependent upon theof the blood coloring-matter from the latter. It is possible Fig. 154.—LtNG, HKUoRmiAcic iNFAurr (Xati«ai. Sur.).1. Pleura. B. SmM infarct centrally placed. The Urjje infarct shows the elevation of the pleura, the hrmorrhajpc Mjffusion of ihc central area, ami the pcri|>hcry of »tor> llVI« • r tt ri Iff Ml< II 284 GENERAL PATHOLOGY. a change occurs, and that the results of the two processes may be thesame; but certain infarcts are clearly anemic from the beginning, andothers are hemorrhagic early in their development. The changes revealed by the histologic examination of the necrotictissue will not be exactly the same in different stages of the are practically those already mentioned when considering coagu-lation necrosis. (See p. 251.) In emboli containing bacteria (infective emboli), and in those con-taining the specific cellular elements of tumors (neoplastic emboli),arrest is followed
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