The malarial fevers, haemoglobinuric fever and the blood protozoa of man . Fig. I.—^Plasmodium vivax. (TertianPlasmodium.) Double infection ofred corpuscle with ring , X 1200. Fig. 2.—Plasmodium vivax. (Tertian plas-modium.) Young amoeboid forms. Photo-micrograph, X 1200. parasite grows older, becoming the schizont, it develops very marked amoeboidactivity, constantly changing its shape, but is still indistinct in outline, requiringvery careful examination to distinguish it in the infected red corpuscle. In the course of from six to eight hours minute granules of a reddish
The malarial fevers, haemoglobinuric fever and the blood protozoa of man . Fig. I.—^Plasmodium vivax. (TertianPlasmodium.) Double infection ofred corpuscle with ring , X 1200. Fig. 2.—Plasmodium vivax. (Tertian plas-modium.) Young amoeboid forms. Photo-micrograph, X 1200. parasite grows older, becoming the schizont, it develops very marked amoeboidactivity, constantly changing its shape, but is still indistinct in outline, requiringvery careful examination to distinguish it in the infected red corpuscle. In the course of from six to eight hours minute granules of a reddish-brown pigment develop within the hyaline cytoplasm and the outline of theorganism becomes more clearly distinguishable. The pigment is of a peculiarreddish-brown color, and in the form of very fine granules arranged irregularlythroughout the protoplasm, and is in very active, dancing motion, producedby currents within the protoplasm. As development proceeds amoeboid THE ETIOLOGY OF THE MALARIAL FEVERS. 21 motion becomes less pronounced, and when the organism is ful
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