. The pathology and differential diagnosis of infectious diseases of animals : prepared for students and practitioners of veterinary medicine . Veterinary medicine; Communicable diseases in animals. NECROBACILLOSIS 227 The metabolic products are exceedingly poisonous. At the point of entrance the organisms multiply, producing a marked reaction of congestion, followed by an exudation rich in fibrin-forming substances and a defensive immigration of leucocytes. The products of the organism acting upon these defensive processes of the body cause their necrosis. With the death of the tissues the ba


. The pathology and differential diagnosis of infectious diseases of animals : prepared for students and practitioners of veterinary medicine . Veterinary medicine; Communicable diseases in animals. NECROBACILLOSIS 227 The metabolic products are exceedingly poisonous. At the point of entrance the organisms multiply, producing a marked reaction of congestion, followed by an exudation rich in fibrin-forming substances and a defensive immigration of leucocytes. The products of the organism acting upon these defensive processes of the body cause their necrosis. With the death of the tissues the bacilli invade the deeper. Fig. 50. areas of necrosis in the liver of a pig due to B. necrophorus. layers. The lesions due to this organism are liable to be found, as already stated, in any organ of the body or on any mucous membrane or the skin. Necrobacillosis of the skin. Necrotic inflammation of the skin or dermatitis gangrenosa forms a group of destructive inflammations of the skin following various causes such as burning, freezing, sHght trauma or pressure during which B. necrophorus may gain entrance to the tissues. Frohner describes an enzootic of this disease in Please note that these images are extracted from scanned page images that may have been digitally enhanced for readability - coloration and appearance of these illustrations may not perfectly resemble the original Moore, Veranus A. (Veranus Alva), 1859-1931. New York : Macmillan


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Keywords: ., bookcentury1900, bookdecade1910, booksubjectveterin, bookyear1916