The Medical clinics of North America . ? Fig. 110.—Liver, showing general destructive lesions and cellular infiltration. neutrophils and eosinophils. In some areas hemorrhage number of cells with extensively vacuolated cytoplasm wereseen, but these appeared to have intact nuclei, and, indeed, atthis stage of the process, it was impossible to make an estimateof the possible relative amount of nuclear damage in contrastto cytoplasmic; fibroblastic reaction was as yet not in evidence(Figs. 110, 111). 45§ WILLIAM J. KERR, GLANVILLE Y. RUSK Lungs were negative. Pancreas showed scattered
The Medical clinics of North America . ? Fig. 110.—Liver, showing general destructive lesions and cellular infiltration. neutrophils and eosinophils. In some areas hemorrhage number of cells with extensively vacuolated cytoplasm wereseen, but these appeared to have intact nuclei, and, indeed, atthis stage of the process, it was impossible to make an estimateof the possible relative amount of nuclear damage in contrastto cytoplasmic; fibroblastic reaction was as yet not in evidence(Figs. 110, 111). 45§ WILLIAM J. KERR, GLANVILLE Y. RUSK Lungs were negative. Pancreas showed scattered foci of postmortem the organ was negative both as to glandular structuresand islands of Langerhans. Adrenals.—No abnormality was found. Stomach appeared Fig. 111.—Liver, showing general destructive lesions and cellular infiltration. Kidneys showed marked parenchymatous degeneration, alsoan occasional small focus of fibrosis and lymphocytic infiltration. That the degree of liver atrophy, as judged by weight, wasnot great in this case, as compared with the often recordedweights, is obvious, but we feel justified in placing the caseunder the caption of acute yellow atrophy in view of the micro-scopic appearance as recorded above. ACUTE YELLOW ATROPHY AND HYPERTHYROIDISM 459 DISCUSSION A certain degree of jaundice is occasionally observed in theterminal stages of thyrotoxicosis. The mechanism of the pro-duction of such jaundice has not been explained. It seems im-probable that it has any relation to cardiac decompression andconsequent passive congestion. It also seems improbable thatit may be due to blood destruction, or at least we know of noevidence to indicate it. That the liver may be at fault, and byan extensive destruction analogo
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