Diseases of the chest and the principles of physical diagnosis . ^ amarked lowering of resistance to material infection. The most common lesion in the lungs is a hroncho-pneumonia. Thebroncho-pneumonia may be caused by the influenza bacillus alone by DISEASES OF THE LUXGS 447 direct extension from the bronchi. More often, however, the pneumo-coccus is responsible for the broncho-pneumonia. The broncho-pneumonicareas are often Hmited to or most marked about the hilus of the observers have emphasized the predilection of the lesions for theperiphery of the lungs and have also directed a
Diseases of the chest and the principles of physical diagnosis . ^ amarked lowering of resistance to material infection. The most common lesion in the lungs is a hroncho-pneumonia. Thebroncho-pneumonia may be caused by the influenza bacillus alone by DISEASES OF THE LUXGS 447 direct extension from the bronchi. More often, however, the pneumo-coccus is responsible for the broncho-pneumonia. The broncho-pneumonicareas are often Hmited to or most marked about the hilus of the observers have emphasized the predilection of the lesions for theperiphery of the lungs and have also directed attention to the hemor-rhagic character of the process. In many cases there is relatively littleconsolidation. The pneumonic areas may consist of small shot-hkenodules or smaller areas may become confluent forming areas the sizeof an EngHsh walnut or larger (see Figs. 284, 285, 286). The colormay be blackish-red, red or grajdsh-red. Even in cases in which con-gestion is not a marked feature and the amount of consolidated tissue. Fig. 287.—Interstitial type of broncho-pneumonia. {Camp Pike Pneumonia Commission.) is relatively sHght, there is often a thin, copious, bloody exudate in thebronchioles and lung tissue which pours out of the nostrils after death. In a certain number of cases the interstitial tissues are involvedconstituting what has been termed an interstitial hroncho-pneumoriia(see Fig. 287). In these cases microscopic examination shows thickeningof the walls of the bronchi and bronchioles and the alveolar walls in theimmediate neighborhood of the bronchioles are also thickened by aninfiltration of mononuclear cells. At one stage the peribronchial in-filtration presents a gross appearance closely resembhng tubercles,and MacCallum suggests that it is possible that these lesions have 448 DISEASES OF THE BROXCHI, LUXGS, PLEURA, AND DIAPHRAGM perhaps been mistaken for tuberculosis when the condition followsmeasles. At a later stage the interlobular septa become greatly t
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Keywords: ., bookcentury1900, bookdecade1920, booksubjectdiagnos, bookyear1920
