A system of surgery . eparated by fine fibre-cells and fibres. As the diseaseprogresses, a tissue like loose granulation tissue is formed, whichtends to become more fibrous; new capillaries are developed in it,which communicate with the vasa vasorum. By this growth ofthe inner coat the lumen of the artery is narrowed, generally dis-torted, often converted into a mere slit; and it may be completelyobliterated in the case of small arteries, either by extension of thegrowth of the intima, or by the formation of a thrombus. Thetunica media is generally unaffected; occasionally it is involved by TU
A system of surgery . eparated by fine fibre-cells and fibres. As the diseaseprogresses, a tissue like loose granulation tissue is formed, whichtends to become more fibrous; new capillaries are developed in it,which communicate with the vasa vasorum. By this growth ofthe inner coat the lumen of the artery is narrowed, generally dis-torted, often converted into a mere slit; and it may be completelyobliterated in the case of small arteries, either by extension of thegrowth of the intima, or by the formation of a thrombus. Thetunica media is generally unaffected; occasionally it is involved by TUBERCULOUS ARTERITIS. 559 an extension of the disease in the intima. The tunica adventitia isgenerally thickened by an addition of fibrous tissue; in rare casesthe change is most marked in the outer coat. The disease is chronic,and probably recurrent; it is the cause of thrombosis and localanaemia, and so leads to cerebral softening and to the degenerativechanges in a gumma; as already mentioned, it is believed by some. Kg. 160.—Syphilitic Arteritis. (From the Path. Soc. Trans., 1887.) a, Outer coat; b, middle coat; c, fenestrated membrane of Henle: internal to which is the greatlythickened intima; d, greatly reduced lumen of the artery. (A specimen of Dr. Sharkeys.) to play an important part in the causation of aneurysm. A prolongedcourse of iodide of potassium is the best means of combating thedisease (Fig. 160). 6. Tuberculous arteritis.—The arteries in tissues the seat oftubercular lesions show a change very similar to that in intima is enormously thickened by a fine cell growth, and theother coats are usually the seat of inflammatory change; but theessential and most constant and marked change is the thickening ofthe intima, which is caused by the tubercle bacilli finding their wayinto the vessel-wall. The lumen of the vessel may be reduced to afine slit only, or be wholly obliterated by the growth of the intima. 7. Obliterating arteritis.—This name has b
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