. Manual of antenatal pathology and hygiene : the foetus. pecialised cell of the gland. Nevertheless no hairs were tobe seen in any of the sections examined. There is no adipose tissue 3;12 ANTENATAL PATHOLOGY AND HYOIENE Id 1)C noted, and the corium presents no striking alterations; hereand there traces of sudoripai-ous glands were visible, but no spiralducts were observed. At certain places, and especially near theterminations of the sebaceous glands, open spaces were noticeable,l)ut I am inclined to regard these as artificially produced during pre-paration for histological examination. Fina
. Manual of antenatal pathology and hygiene : the foetus. pecialised cell of the gland. Nevertheless no hairs were tobe seen in any of the sections examined. There is no adipose tissue 3;12 ANTENATAL PATHOLOGY AND HYOIENE Id 1)C noted, and the corium presents no striking alterations; hereand there traces of sudoripai-ous glands were visible, but no spiralducts were observed. At certain places, and especially near theterminations of the sebaceous glands, open spaces were noticeable,l)ut I am inclined to regard these as artificially produced during pre-paration for histological examination. Finally, there was no centralrod of cartilage, and the vascularity of the tumour was little marked ;there was no pigmentation. The congenital growth in this case consisted, as has been shown,of skin; but it has to be noted that in certain particulars the skinwas in an imperfectly developed state. There were no hairs, althoughthe tumour took its origin from a scalp well supplied with hair; thesweat glands were only represented by traces; the sebaceous glands. were present in every stage from the most rudimentary to the fullyformed; and there was a total absence of adipose tissue in tlie sub-cutaneous layer. The outstanding feature was the hyperplasia of theprickle-cell layer. The first impression gained from the study of thehistology of the growth was that here we had to do with tissueswhich had fallen behind in the general development of the second notion was that some source of irritation must also havebeen in action, for, as Dr. Allan Jamieson (wlio was kind enough toexamine the sections witli me, and advise me thereupon) pointeil out,the appearances, especially in the Malpighian layer, closely resembledthose seen in some forms of chronic eczema. These ideas, along witha similarity in tlie appeai-ance and history of the growth, led me again a preauricular ap})endage wliich I removed in 1894from a boy, 12 years of age. Although smaller in size, it resembled ACA
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