. Diseases of the heart and thoracic aorta. Fig. 228. Fig. 221 .—Constriction of the pulmonary orifice. {After Peacock.) The pulmonarj valves are united so as to form an infundibular or funnel-shapeopening from the ventricle into the artery; the ductus arteriosiis is pervious, andthe foramen ovale open. Fig. 228.—For description see page 545. Course of the Blood in the Foetus. 545 pulmonary circulation is imperfectly supplied with blood ;the tricuspid may become incompetent, and the systemicvenous system engorged. Compensation in cases of thisdescription is chiefly effected by the hypertrophy
. Diseases of the heart and thoracic aorta. Fig. 228. Fig. 221 .—Constriction of the pulmonary orifice. {After Peacock.) The pulmonarj valves are united so as to form an infundibular or funnel-shapeopening from the ventricle into the artery; the ductus arteriosiis is pervious, andthe foramen ovale open. Fig. 228.—For description see page 545. Course of the Blood in the Foetus. 545 pulmonary circulation is imperfectly supplied with blood ;the tricuspid may become incompetent, and the systemicvenous system engorged. Compensation in cases of thisdescription is chiefly effected by the hypertrophy of the rightventricle. In congenital cases, the secondary results, and the mannerin which compensation is established, vary in different cases,and depend chiefly upon the period of intra-uterine life atwhich the stenosis of the pulmonary artery is established. But in order that these points may be thoroughly under-stood, it is necessary to remember the course of the circulationduring intra-uterine life. It is represented in fig. 229, a
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Keywords: ., bookcentury1800, bookdecade1880, booksubjectheart, bookyear1884
