Manual of pathology : including bacteriology, the technic of postmortems, and methods of pathologic research . occasionally choles-terin. Degenerationmay spread to and in-vade the media, al-though extensive in-volvement of thisstratum is not . celkilar infiltrationof the arterial wall maybe first manifest or mostmarked a r o xi n d thebranches of the vasavasorum. Atheroma gives riseto loss of elasticity,weakening of the wall,and increased liabilityto aneurysm. Thearterial tube being maderigid, the cardiac work isthought to be increased,although upon this pointauthorities are not full
Manual of pathology : including bacteriology, the technic of postmortems, and methods of pathologic research . occasionally choles-terin. Degenerationmay spread to and in-vade the media, al-though extensive in-volvement of thisstratum is not . celkilar infiltrationof the arterial wall maybe first manifest or mostmarked a r o xi n d thebranches of the vasavasorum. Atheroma gives riseto loss of elasticity,weakening of the wall,and increased liabilityto aneurysm. Thearterial tube being maderigid, the cardiac work isthought to be increased,although upon this pointauthorities are not fullyagreed. II. Arteritis obliter-ans^ is observed in asso-ciation with tertiarysyphilis and contractingkidney, and is present, asa local process, in thevessels supplying areasof tuberculosis. Obliter-ative changes are physiologic in the vessels of the fetus which become 1 Bradford and Lawrence, Jour, of Path, and Bact., May, iSgS, p. , Lancet, March 15, 1902. Morgan, Jour. Amer. Med. Assoc, , 1902, p. 1359. Branson, Path. Soc. of London, Dec. 6, 1904. Whitacre,N. Y. Med. Jour., Jan. 28, Fig. 259.—.\oRTA, Opened, Showing Different Types of .\th- EROMA. The surface is most extensively altered by infiltration, degeneration, andnecrosis. Many of the necrotic areas are calcified and could befractured by bending. A, A, A. Elevated obstructing patches ofatheroma surrounding exit points of small branches. B, B. Linearatheroma. VASCULAR SYSTEM. 523 useless at birth, and in the arterial supply to the parturient uterus, afterlabor. A similar, but not identical process occurs in the branches ofligated vessels, especially those not i)articipatin},f in the production ofany succeeding collateral anastomosis. Morbid A)iato»iy.—The disease is y)rimarily an alTection of thesmaller vessels, but may extend centripetally, producing thrombosis inthe larger trunks. The affected vessels are hard, often cord-like, usuallysmooth externally, although the obliterating 7)rocess is
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