. Medical diagnosis for the student and practitioner. Fig. 522.—(3) Separate globoid bodies. X 1000. (4) Aggregated masses of globoidbodies. X 1000. (5) Chains and pairs of globoid bodies. X 1000. (6) Chains of globoidbodies compared with streptococcus pyogenes. (7) Agar fragment showing pairs of globoidbodies compared with streptococcus pyogenes. X 1000. (Flexner and Noguchi, Journalof Experimental Medicine, Vol. XVIII, 1913.) neurons affected, axis cylinders, nerve roots, peripheral nerves and themuscles activated by them. The chief pathologic change is one of peri-vascular lymphangitic infl
. Medical diagnosis for the student and practitioner. Fig. 522.—(3) Separate globoid bodies. X 1000. (4) Aggregated masses of globoidbodies. X 1000. (5) Chains and pairs of globoid bodies. X 1000. (6) Chains of globoidbodies compared with streptococcus pyogenes. (7) Agar fragment showing pairs of globoidbodies compared with streptococcus pyogenes. X 1000. (Flexner and Noguchi, Journalof Experimental Medicine, Vol. XVIII, 1913.) neurons affected, axis cylinders, nerve roots, peripheral nerves and themuscles activated by them. The chief pathologic change is one of peri-vascular lymphangitic inflammation and round-cell infiltration about thecentral arteries each of which supplies a segment of the cord about 5 cm. inlength, which corresponds to the focal nature of the changes noted in thedisease. ACUTE INFECTIOUS POLIOMYELITIS IO9I I I **k
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