Manual of pathological anatomy . ed, and sometimes cavities, with smooth internalsurface, are formed partly, or wholly, by the enlarged contain purulent mucus, often very offensive and condition usually affects one lung only, and sometimes onlyone lobe. The above description applies to the most advancedstages of the disease. Early stages show little more than indura-tion, and are not always easily recognized w^ith the naked eye,though microscopic examination may show the characteristicthickening of the alveolar walls. The histology of a lung in this 490 CHRONIC PNEUM
Manual of pathological anatomy . ed, and sometimes cavities, with smooth internalsurface, are formed partly, or wholly, by the enlarged contain purulent mucus, often very offensive and condition usually affects one lung only, and sometimes onlyone lobe. The above description applies to the most advancedstages of the disease. Early stages show little more than indura-tion, and are not always easily recognized w^ith the naked eye,though microscopic examination may show the characteristicthickening of the alveolar walls. The histology of a lung in this 490 CHRONIC PNEUMONIA. condition is represented in Fig. 108. The walls are seen greatlythickened, and containing pigment granules, while the air cellsare filled with large irregular masses resembling mucus corpuscles,but often of abnormal size, as if several were fused together, andalso containing pigment. It is not easy to say whether thesebodies are produced where they are found, or haye entered theair cells from the bronchial tubes. Fig. Section of an indurated lung affected with chronic pneumonia, showing- the wallsof the alveoli very greatly thickened, and the cavities filled with mucous corpusclesand larger masses containing pigment. We do not, however, always find that chronic pneumonia con-sists in changes of this kind. It may, on the other hand, resembleacute lobar pneumonia in having the air cells filled with fibrinousexudation, and the only peculiarity observable in the exudationis the preponderance of tough, opaque granular fibrin and thesmall number of cells. In other cases we have found a remarkablyluxuriant development of epithelial structures, which have quitefilled the alveoli. Induration and contraction, howev(;r, in the enddepend on the formation of a fibroid (now called lymphadenoidtissue) and gradual obliteration or replacement of the pulmonarytissue by it. This tissue is represented in Fig. 109. It varies indifferent cases in the proportions of the lymphoid cells and
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