Clinical electrocardiography . as to submergethe sinus rate and establish flutter. That such functional pathologyexists, however, is evident. We have no evidence that fluttercan be purely of neurogenic origin. The electrocardiograms of flutter are characterized by theextremely rapid auricular rate, more than 200 per minute, and byinversion of the auricular P wave. Partial heart-block is usuallypresent, the auriculoventricular ratio varying from 2:1 to 5 : 1;occasionally paroxysm of 1:1 association occurs, and again,complete heart-block may be present. Figure 51 illustrates the mechanism in flu
Clinical electrocardiography . as to submergethe sinus rate and establish flutter. That such functional pathologyexists, however, is evident. We have no evidence that fluttercan be purely of neurogenic origin. The electrocardiograms of flutter are characterized by theextremely rapid auricular rate, more than 200 per minute, and byinversion of the auricular P wave. Partial heart-block is usuallypresent, the auriculoventricular ratio varying from 2:1 to 5 : 1;occasionally paroxysm of 1:1 association occurs, and again,complete heart-block may be present. Figure 51 illustrates the mechanism in flutter. Figure 52shows a paroxysm of 1 : 1 flutter; Fig. 53, 2 : 1 flutter; Fig. 54,3 : 1 flutter; Fig. 55: 4:1 flutter; Fig. 56, 5 : 1 flutter; Fig. 57shows flutter with complete heart-block. Flutter may be conveniently classified as paroxysmal or chronic,depending on the duration of the disorder. We use the term ECTOPIC RHYTHMS AND TACHYCARDIAS 83 III I III I I III I I I I MlUH, Fig. 51.—Schematic graph of auricular Fig. 52.—Auricular flutter, 1 : 1 rhythm. Rate 232. Electrocardiogram in Derivation II.
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