. The practice of medicine; a text-book for practitioners and students, with special reference to diagnosis and treatment . Fig. 144.—Diagram of Lesion Showing Brown-Sequards Paralysis—{after Slarr). L. Lesion in left half of cord cuts off motor im-pulses to left leg, sensory impulses from right leg,and sensory impulses from eleventh dorsal Fig. 145.—Schema Showing ChiefSymptoms in Left Unilateral Lesionof the Dorsal Cord—[after Erb). Oblique shading at a signifies motorand vasomotor paralysis; verticalshading cutaneous anesthesia at band d\ dots on a cutaneous hyper-esthesia, b. Small
. The practice of medicine; a text-book for practitioners and students, with special reference to diagnosis and treatment . Fig. 144.—Diagram of Lesion Showing Brown-Sequards Paralysis—{after Slarr). L. Lesion in left half of cord cuts off motor im-pulses to left leg, sensory impulses from right leg,and sensory impulses from eleventh dorsal Fig. 145.—Schema Showing ChiefSymptoms in Left Unilateral Lesionof the Dorsal Cord—[after Erb). Oblique shading at a signifies motorand vasomotor paralysis; verticalshading cutaneous anesthesia at band d\ dots on a cutaneous hyper-esthesia, b. Small anesthetic Small hyperesthetic zone. extend to the bladder and rectum. After complete or nearl} completesection the muscles are usually flaccid and the deep reflexes absent. Thereis no rapidly developing atrophy, and the muscles respond normally toelectricity. No satisfactory explanation has as yet been offered of theabolition of the deep reflexes in complete or nearly complete transverselesion of the cord above the level of the reflex arcs; although neuritis issupposed by some to be the cause of this loss of the deep reflexes, it isprobably not the cause in all cases. Second, there is impaired sensibilityin the parts supplied by sensory ner\es associated with correspondingsegments below the lesion. Anesthesia does not, however
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