Clinical tuberculosis . ation.—Aside from the pathological changesAvhich are caused directly by the bacillary process there is aninllannnation of greater or lesser extent and severity affectingthe tissues adjacent to the tubercles. This collateral inflamma-tion depends upon the virulence and number of bacilli on theone hand and the receptivity of the tissues on the other. Itmay be a productive inflammation or a degenerative exudation may be serous, fibrinous, cellular, sanguinous, orpurulent. It may undergo the same changes as the tubercleitself, producing new connective , o
Clinical tuberculosis . ation.—Aside from the pathological changesAvhich are caused directly by the bacillary process there is aninllannnation of greater or lesser extent and severity affectingthe tissues adjacent to the tubercles. This collateral inflamma-tion depends upon the virulence and number of bacilli on theone hand and the receptivity of the tissues on the other. Itmay be a productive inflammation or a degenerative exudation may be serous, fibrinous, cellular, sanguinous, orpurulent. It may undergo the same changes as the tubercleitself, producing new connective , or softening. Former-ly it was thought that these changes were due to other bacteria,so-called mixed infections; but now it is known that all thesechanges can be produced -without the help of any other organ-ism than the tubercle bacillus. i^;;i^€^*i^v • N V Ml .?**, , % Fig. 1.—Schematic illustration ol a tubercle. Center, necrotic giant cell; second layer,epithelioid cells; outer layer, lymphocytes. (Adanii.). Fig. -Illustrating conversion of tubercle into scar tissue. Tubercle undergoing fibrosis;center partially fibrous. (Fowler and Godley.) ?=i2-^-s»^.clinicaltubercul01pott
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Keywords: ., bookcentury1900, bookdecade1920, bookpublisherstlou, bookyear1922
