AMAarchives of neurology & psychiatry . chloric acid. The total aciditv was 33. —FrXICULAR MYELOPATHY 369 Course of Illness.—During her stay in the hospital, the patient developedmental disturbance. She became disoriented, speech was incoherent at times,and there was a mild muttering delirium. Definite lateral nystagmus appearedin both eyes, and ataxia of the left arm developed. The knee reflexes were lost. Pathologic Anatomy. — Material and Methods: The spinal cords of thepatients in Cases 1 and 2. and the brain in Case 1, were available for blocks were fixed in
AMAarchives of neurology & psychiatry . chloric acid. The total aciditv was 33. —FrXICULAR MYELOPATHY 369 Course of Illness.—During her stay in the hospital, the patient developedmental disturbance. She became disoriented, speech was incoherent at times,and there was a mild muttering delirium. Definite lateral nystagmus appearedin both eyes, and ataxia of the left arm developed. The knee reflexes were lost. Pathologic Anatomy. — Material and Methods: The spinal cords of thepatients in Cases 1 and 2. and the brain in Case 1, were available for blocks were fixed in liquor formaldehydi, alcohol and Weigerts gliamordant. Some formaldehyd fixed material was further carried into Miillersfluid and into Miiller-osmic-acid solution. Celloidin, paraffin and frozen sec-tions were made. The following stains were used: Delafields hematoxylin,Heidenhains iron hematoxylin, Spielmeyers myelin sheath stain, Weigert-Pal,Marchi, Herxheimers fat stain, Bielschowskys silver, Alzheimer-Mann andJacobs Fig. 1 (Case 1).—Section of a dorsal segment showing the almost completeinvolvement of the dorsal, lateral and anterior pyramidal tracts. Gross Anatomy: Both spinal cords appeared markedly reduced in meninges presented no evidence of gross pathologic change. Several crosscuts made in the cord showed a distinct degenerative process in the dorsal,lateral and ventral columns. In Case 1 the lateral and anterior pyramidaltracts showed a most intense destructive process in the lumbar region. Inthe cervical segments a destructive process in the region of the spinocerebellartracts was added. The areas involved were best brought out in stainedpreparations, presenting a picture of distinct combined funicular disease(Fig. 1). A section in the dorsal region, stained by the Marchi method, showedby the appearance of large areas staining black that the process was still Anatomy.—In studying the diseased areas in the spinal cord,
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