General and dental pathology with special reference to etiology and pathologic anatomy; a treatise for students and practitioners . han two millimeters inaverage depth. The peridental membrane, alveolar process, andoverlying gum tissue are destroyed to the level of the salivarydeposit as the result of a combination of pressure atrophy andbacterial activity. Consequently, in the salivary calculi form ofpyorrhea alveolaris (correctly interpreted, the process shouldbe termed calcic gingivitis, pericementitis and alveolitis), theformation of pockets does not occur, and the degree of suppurativeinf


General and dental pathology with special reference to etiology and pathologic anatomy; a treatise for students and practitioners . han two millimeters inaverage depth. The peridental membrane, alveolar process, andoverlying gum tissue are destroyed to the level of the salivarydeposit as the result of a combination of pressure atrophy andbacterial activity. Consequently, in the salivary calculi form ofpyorrhea alveolaris (correctly interpreted, the process shouldbe termed calcic gingivitis, pericementitis and alveolitis), theformation of pockets does not occur, and the degree of suppurativeinflammation under the deposit, if there should be any, is not pro-nounced at any time. Pus forms at a very slow rate, and it isseen at the junction of the deposit with the soft tissues, or uponthe removal of the deposit. Pus is not by any means invariablyassociated with salivary calculi. In those cases in which it ispresent, the suppurative inflammation, in addition to the phe-nomena of pressure atrophy, is responsible for the destructionof the investing tissues; while in those cases in which no evidence PYOKKIIKA UAKOPAPIK 475. Fig. 370.—Subgingival deposits on the rootsurfaces of the lower central and lateral of the alveolar process and peri-dental membrane on the approximal surfaces ofthese teeth, resulting in the formation of so-called pyorrhea pockets. Fig. 371.—Absence of approxi-mal contact accounting for the for-mation of a pocket between lateralincisor and cuspid.


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Keywords: ., bookcentury1900, bookdecade1920, booksubjectpathology, bookyear19