Clinical electrocardiography . Fig. 21.—Sinus arhythmia. Electrocardiogram in Derivations I, II, and III. slowing of the entire heart. Sinus arhythmia occurs in individualswhose vagus mechanism is temporarily or permanently unstable,and in whom nervous irritability permits otherwise inadequatestimuli to act. Exertion frequently induces sinus arhythmia. 56 CLINICAL ELECTROCARDIOGRAPHY It must be remembered, however, that sinus arhythmia, per se,while not indicative of organic heart disease, may be associatedwith changes in the heart of a very serious nature. Sinus arhythmiain older patients, wi
Clinical electrocardiography . Fig. 21.—Sinus arhythmia. Electrocardiogram in Derivations I, II, and III. slowing of the entire heart. Sinus arhythmia occurs in individualswhose vagus mechanism is temporarily or permanently unstable,and in whom nervous irritability permits otherwise inadequatestimuli to act. Exertion frequently induces sinus arhythmia. 56 CLINICAL ELECTROCARDIOGRAPHY It must be remembered, however, that sinus arhythmia, per se,while not indicative of organic heart disease, may be associatedwith changes in the heart of a very serious nature. Sinus arhythmiain older patients, with evident degenerative myocardial changes,usually is associated with a persistently slow pulse-rate. Figure20 represents the character of the arhythmia. Figure 21 is anelectrocardiogram of a patient with sinus arhythmia. All com-plexes are normal in contour and maintain their proper relationshipto one another. The arhythmia occurs through variations in thelengths of the cycles resulting from acceleration and slowing ofrate.
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