Practical pathology; a manual for students and practitioners . um is invariably present. 302. In a second form of suppurative nephritis, frequentlyassociated with ulcerative endocarditis, there can be no doubt that themultiple abscesses result from the impaction of septic emboli in theinterlobular vessels, and in the intertubular and intra-glomerularcapillaries. These emboli consist of scraps of fibrinous lymph inwhich are embedded micrococci, which latter multiply rapidly andset up abscess formation as already described (i^ 229), as, in additionto the condition above described, masses of deep
Practical pathology; a manual for students and practitioners . um is invariably present. 302. In a second form of suppurative nephritis, frequentlyassociated with ulcerative endocarditis, there can be no doubt that themultiple abscesses result from the impaction of septic emboli in theinterlobular vessels, and in the intertubular and intra-glomerularcapillaries. These emboli consist of scraps of fibrinous lymph inwhich are embedded micrococci, which latter multiply rapidly andset up abscess formation as already described (i^ 229), as, in additionto the condition above described, masses of deeply stained micrococcimay be found in all these positions (in a section stained withmethylanilin-violet). Under certain conditions nephritic abscesses become more chronic,in which case large caseous masses, encapsuled by firm fibrous tissue,may be met with. On microscopic examination, the tissue around SUPPURA TT VE NEPHRl TIE JNFA R C TION 423 is found to be atrophied, whilst chronic interstitial inflammatorychanges are also met with. (See >5 298 et seq.). \jA^^*m
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