American journal of physiology . to fe L. J. J. Muskens. second. The ventricle then loses one beat,while only a slight elevation in the auricularcurve gives evidence of the systole of theauricle. In the, next cycle also the ventricledoes not beat, but the vagus influence is al-ready waning, for the force of auricular systoleis observed to increase. In the next cyclethe ventricle itself contracts again, the auriculo-ventricular interval is still prolonged, being isec. Gradually the normal interval and thenormal force of auricular systole is restored. Under still more powerful vagus excitati


American journal of physiology . to fe L. J. J. Muskens. second. The ventricle then loses one beat,while only a slight elevation in the auricularcurve gives evidence of the systole of theauricle. In the, next cycle also the ventricledoes not beat, but the vagus influence is al-ready waning, for the force of auricular systoleis observed to increase. In the next cyclethe ventricle itself contracts again, the auriculo-ventricular interval is still prolonged, being isec. Gradually the normal interval and thenormal force of auricular systole is restored. Under still more powerful vagus excitationthe auricular systoles become too small to berecorded. The first cycle after the so-calledarrest is in such cases often incomplete. Theventricle beats, but the auricular contractionremains invisible, appearing first in the nextcycle; or the auricular contractions becomevisible again, while the ventricular contractionsdo not yet appear. The effect of powerfulvagus excitation on the auriculo-ventricularinterval is shown in the a


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