A non-surgical treatise on diseases of the prostate gland and adnexa . ous. This disease of the gland is one of passive ve-nous congestion, soft submucous infiltration and a swollenenlargement of the organ, which generally results in in-flammation not only of the gland itself, but of most allthe other pelvic viscera. It is somewhat analogous to thecongested and inflamed condition of the uterus and itsappendages. Not every woman that has venous stasis andan inflamed womb has fil^roid tumors developed within itswails. In fact, the latter is rare as compared with thenumber of cases of the former.
A non-surgical treatise on diseases of the prostate gland and adnexa . ous. This disease of the gland is one of passive ve-nous congestion, soft submucous infiltration and a swollenenlargement of the organ, which generally results in in-flammation not only of the gland itself, but of most allthe other pelvic viscera. It is somewhat analogous to thecongested and inflamed condition of the uterus and itsappendages. Not every woman that has venous stasis andan inflamed womb has fil^roid tumors developed within itswails. In fact, the latter is rare as compared with thenumber of cases of the former. In like manner fibrinoustumors, or hypertrophy of the prostate, is rare as comparedwith the numerous occurrences of congested enlargement.• I have treated quite a number of cases of this class ofprostatitis, that had been treated by different physiciansfor many years, and pronounced hypertrophy and incura-ble. Some of these cases had not only been treated by thefamily physicians, but by many of the leading specialistsin this line. 75 76 PROSTATE GLAND AND Fig. XII. ENLARGEMENT OF THE PROSTATE. 77 It is somewhat difficult in some cases to diflEerentiate atfirst between congested enlargement and fibrinous indu-rated hypertroph5^ The diagnosis will be considered moreat length under the head of diagnosis that will follow uponthis subject. I shall add here, however, that, basing astatement upon my own clinical experience, at least twentycases of congested enlargement of the gland occur to oneof hypertrophy. I do not include in this estimate chronicprostatic folliculitis common to young men. Cause.—The most potent etiological factors, productiveof this form of disease of the prostate, are common withthose that excite chronic inflammation of the gland as de-tailed in Chapter III. When folliculitis is not arrested andit is permitted to extend and induce parechymatous pros-tatitis and plastic exudation, interstitial infiltration is thenatural sequence. The exudate, thus pr
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