. Contributions to the genetics of Drosophila melanogaster. Drosophila melanogaster; Heredity; Karyokinesis. 346 GENES MODIFYING NOTCH. Half of the daughters are normal, half are heterozygous Notch. The normal daughters and normal sons never transmit the Notch gene, which, therefore, never gets into the male hne or into the line of normal Fig. 91. Dexter obtained his mutant in a cross between beaded and wild. The Notch that I used arose independently in descendants of ves- tigial flies, in which stock the factor may have already existed. This mutant has, however, originated several
. Contributions to the genetics of Drosophila melanogaster. Drosophila melanogaster; Heredity; Karyokinesis. 346 GENES MODIFYING NOTCH. Half of the daughters are normal, half are heterozygous Notch. The normal daughters and normal sons never transmit the Notch gene, which, therefore, never gets into the male hne or into the line of normal Fig. 91. Dexter obtained his mutant in a cross between beaded and wild. The Notch that I used arose independently in descendants of ves- tigial flies, in which stock the factor may have already existed. This mutant has, however, originated several times in other cultures in the laboratory. It is by no means one of the rarer mutations. VARIATION OF NOTCH. The most conspicuous character of the female heterozygous for Notch is the serration at the end of the wings (fig. 91) caused by the absence of the marginal bristles and generally accompanied by a slight. Please note that these images are extracted from scanned page images that may have been digitally enhanced for readability - coloration and appearance of these illustrations may not perfectly resemble the original Carnegie Institution of Washington; Morgan, Thomas Hunt, 1866-1945; Bridges, Calvin B. (Calvin Blackman), 1889-1938; Sturtevant, A. H. (Alfred Henry), 1891-1970. Washington, Carnegie Institution of Washington
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