. The chemistry of the injured cell. Cytochemistry; Pathology, Cellular. 118 The Chemistry of the Injured Cell antergan). Histamine appears to exert its effects within a minute or two of injury and continues to dilate capillaries and increase their permeability for at least one to two hours. It is postulated that at the same time as histamine is released, an adrenaline-like substance is brought into contact with the enzyme monoamine oxidase which destroys it. This reaction may be prevented by administration of monoamine oxidase inhibitors. The effects of these inhibitors suggest that the hypot
. The chemistry of the injured cell. Cytochemistry; Pathology, Cellular. 118 The Chemistry of the Injured Cell antergan). Histamine appears to exert its effects within a minute or two of injury and continues to dilate capillaries and increase their permeability for at least one to two hours. It is postulated that at the same time as histamine is released, an adrenaline-like substance is brought into contact with the enzyme monoamine oxidase which destroys it. This reaction may be prevented by administration of monoamine oxidase inhibitors. The effects of these inhibitors suggest that the hypothetical de- struction following injury of an adrenaline-like substance leads to dilatation and increased permeability of small vessels that begins within a few minutes and may last for 24 hours or even longer. Soon after these initial events there is activation of globulins and peptides that increase capillary permeability. The mechanism is unknown but may involve the action of enzymes of the esterase- LOCAL INJURY. Histamine Rel e a s e Activation of Proteases Activation of Serum Globulin. Please note that these images are extracted from scanned page images that may have been digitally enhanced for readability - coloration and appearance of these illustrations may not perfectly resemble the original Cameron, Roy, Sir, 1899-1967; Spector, W. G. (Walter Graham). Springfield, Ill. , Thomas
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