. A text-book of medicine for students and practitioners . eration, but a moderate loss of fibers is sometimes found intiie region of the columns of Goll. Further observations and investigations must teach us whether, as isprobal)le, we shall find closer relations and transition forms between neuroticand spinal atrophy; but meanwhile the disease just described is a form ofprogressive muscular atrophy so well characterized and so readily recognizablein its clinical aspect that a separate description of it is undoubtedly war-ranted. [Dejerine and Sottas have described, under the name of hypertro
. A text-book of medicine for students and practitioners . eration, but a moderate loss of fibers is sometimes found intiie region of the columns of Goll. Further observations and investigations must teach us whether, as isprobal)le, we shall find closer relations and transition forms between neuroticand spinal atrophy; but meanwhile the disease just described is a form ofprogressive muscular atrophy so well characterized and so readily recognizablein its clinical aspect that a separate description of it is undoubtedly war-ranted. [Dejerine and Sottas have described, under the name of hypertrophicinterstitial neuritis, an affection similar to neurotic muscular atrophy, whichalso begins in early life and may affect several members of the same atrophy usually begins in the legs, being most marked at the extremityand gradually extending upward toward the trunk. The arms, and sometimesthe face, are affected later in the same way. The reaction of degeneration canbe found in the affected muscles. There is a very pronounced enlargement of. Fig. 177.—Peroneal muscular atrophy. Legs of tliebrother of the patient shown in Fig. 176. PRIMARY DEGENERATIONS OF THE MOTOR TRACT 383 the nerve trunks, which can often be felt and sometimes even seen. This isthe characteristic feature of the disease. In addition tiiere may be well-marked kyphoscoliosis, loss of tendon reflexes, inability to stand with theeyes closed, Argyll Eobertsoii ])U]»ils, or sluggisli pupils with myosis, hypses-thesia and delayed sensation, very severe shooting pains, ataxia, and occa-sionally intention tremor, scanning speech, and slight exophthahnus. Thesphincters are never involved. The trouble is very slowly progressive. Au-topsies have shown, besides a very extensive and pronounced neuritis, lesionsin the anterior cornua and posterior columns of the cord. The prognosis isunfavorable, and the treatment is like that of other forms of atrophy.—K.] 5. PROGRESSIVE MUSCULAR DYSTROPHY {The MyoiHithi
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