. The pathology and differential diagnosis of infectious diseases of animals : prepared for students and practitioners of veterinary medicine . Veterinary medicine; Communicable diseases in animals. INFECTIOUS ENTERO-HEPATITIS 319 delicate tissues of the young are best adapted for the temporary habitat and rapid multiphcation of this parasite.* The primary seat of the disease is the ceca. From these the hver is secondarily infested through metastasis. Other organs have not been found to be attacked. Hadley refers to lesions in the spleen, kidneys, pancreas, lungs, heart and oviduct. The lesion
. The pathology and differential diagnosis of infectious diseases of animals : prepared for students and practitioners of veterinary medicine . Veterinary medicine; Communicable diseases in animals. INFECTIOUS ENTERO-HEPATITIS 319 delicate tissues of the young are best adapted for the temporary habitat and rapid multiphcation of this parasite.* The primary seat of the disease is the ceca. From these the hver is secondarily infested through metastasis. Other organs have not been found to be attacked. Hadley refers to lesions in the spleen, kidneys, pancreas, lungs, heart and oviduct. The lesions of the ceca are in substance a thickening of the wall, followed in most cases by a destruction of the epithelium and deeper portions of the mucous membrane. This destruction results in the outpouring of a coagulable fluid into the tube. The thickening of the wall may vary considerably in extent from case to case. It may be uniform over the greater por- tion of the tube or it may be limited to circumscribed patches. The commonest seat of these lesions is near the blind end of the tube where it evidently starts and whence it spreads to other portions. Not infrequently only one cecum is diseased, the other remaining normal. The affection of the cecum is due pri- marily to the multiplication of the micro- organism which may take place chiefly either in the mucous membrane, or in the submucous tissue; it may, though rarely, extend into the muscular coat. The thick- ening of the wall is the result of several processes—the multiplication of the para- sites, the increase of the normal tissue ele- ments and later on the accumulation of masses of small cells and some giant cells. In the early stages of the invasion, the adenoid tissue between the tubules and in the submucosa becomes greatly increased, owing to the presence of large numbers of micro-parasites of round or slightly oval outline and from 6 to lOt^ in diameter which stimu- *In this regard it simply follows the rule observ
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Keywords: ., bookcentury1900, bookdecade1910, booksubjectveterin, bookyear1916
