. The chemistry of the injured cell. Cytochemistry; Pathology, Cellular. Acetyl CoA + Acetyl CoA [CH3COC0A + CH3COC0A] Cholestrol Synthesis Acetoacetate [CHoC0-CH2C00H 2CoA 2CoA]. Acetone [CH3COCH3] /3 hydroxybutyrate [CH3CHOH -CH2C00H] Fig. 8. The Formation of Ketone Bodies and Excess Cholesterol in Diabetes. Another possible result of the disturbance is that acetyl CoA is diverted to another reaction in which it is involved, namely, choles- terol synthesis, thus leading to hypercholesterolaemia and perhaps to its deposition in blood vessels (atheroma) . Both these abnormali- ties are common


. The chemistry of the injured cell. Cytochemistry; Pathology, Cellular. Acetyl CoA + Acetyl CoA [CH3COC0A + CH3COC0A] Cholestrol Synthesis Acetoacetate [CHoC0-CH2C00H 2CoA 2CoA]. Acetone [CH3COCH3] /3 hydroxybutyrate [CH3CHOH -CH2C00H] Fig. 8. The Formation of Ketone Bodies and Excess Cholesterol in Diabetes. Another possible result of the disturbance is that acetyl CoA is diverted to another reaction in which it is involved, namely, choles- terol synthesis, thus leading to hypercholesterolaemia and perhaps to its deposition in blood vessels (atheroma) . Both these abnormali- ties are common complications of diabetes. EXOGENOUS POISONS Well documented examples in this group are scarce. The anti- cholinesterases, DFP, parathion, mipafox, apiol produce severe neurological symptoms accompanied by demyelination and death. Their only major common property appears to be inhibition of certain carbonic esterases notably cholinesterase, and esterase linked proteases, chymotrypsin, but the connection between their effect on these enzymes and their toxic properties is uncertain. The opposite effect, that of a specialised enzyme producing in-. Please note that these images are extracted from scanned page images that may have been digitally enhanced for readability - coloration and appearance of these illustrations may not perfectly resemble the original Cameron, Roy, Sir, 1899-1967; Spector, W. G. (Walter Graham). Springfield, Ill. , Thomas


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Keywords: ., bookcentury1900, bookcollectionbio, booksubjectpathologycellular