Manual of pathology : including bacteriology, the technic of postmortems, and methods of pathologic research . e substance. If prepared by Marchis method (). microscopic sections show areas of fatty change. The axis- Hoist. ?? Xorsk Majj. for L:rjxevirlenskal)en, Oct., Tropenhyg., 1901 viii V^ >-> r)riJan., 1904, p. 112. Fajardo. in ? li iiir Arch. f. •f yg. S92 SPECIAL PATHOLOGY. cylinders are usually interrupted at the points where the myelin isdivided. The nuclei of the nerve-sheath increase in number. Gradu-ally, the myelin further degenerates, and, in
Manual of pathology : including bacteriology, the technic of postmortems, and methods of pathologic research . e substance. If prepared by Marchis method (). microscopic sections show areas of fatty change. The axis- Hoist. ?? Xorsk Majj. for L:rjxevirlenskal)en, Oct., Tropenhyg., 1901 viii V^ >-> r)riJan., 1904, p. 112. Fajardo. in ? li iiir Arch. f. •f yg. S92 SPECIAL PATHOLOGY. cylinders are usually interrupted at the points where the myelin isdivided. The nuclei of the nerve-sheath increase in number. Gradu-ally, the myelin further degenerates, and, in many parts of the nerve-tibers, entirely disappears; when this occurs, the primitive sheathcollapses, and ultimately the entire nerve may be destroyed, beingreplaced by fibrous tissue. As the process becomes older compoundgranule cells make their appearance within and around the fibers. Ifthe inflammatory phenomena be slight, the nerve-fibers do not suffer toso great a degree. In the chronic forms the fibrous tissue is usuallygreatly increased; this condition is commonly observed in the peripheral. Fig. 423.—Chronic Interstitial Neuritis, Showing Degeneration in Some of the Nerve-fibers.— {Gordon.)The interstitial tissue is everywhere increased and the perineurium thickened. The patient had arteriosclerosis. nerves particularly of the aged, and is probably the result of arterio-sclerosis. Whether the neuritis be primary or secondary the similar alterationsare observed within the nerve-fibers involved. Degenerative changesoccur in the muscles supplied by the diseased nerves. The fibers losetheir striation, become granular, fragmented and eventually fatty; thenuclei of the sarcolemma and interstitial tissue increase in number, andfinally absorption of the degenerated and necrotic elements leaves theaffected muscles extremely wasted. The condition constitutes oneform of secondar}^ degeneration of muscle (p. 788). Degenerative changes in the nerve-trunk follow various spinala
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