Gynecology . ective tissue. Thehyalin colorless masses then roll together in cloud-like convolutions and formthe corpus albicans. This process of regression occupies about four corpus albicans may remain a long time, but the hyalin material is event-ually entirely absorbed and all trace of the former corpus luteum disappears,excepting the scarred indentation on the surface of the ovary. The size of the corpus luteum varies considerably, reaching sometimes even 40 GYNECOLOGY in non-pregnant women a diameter of 2 cm. If pregnancy occurs its develop-ment is more pronounced, reaching its
Gynecology . ective tissue. Thehyalin colorless masses then roll together in cloud-like convolutions and formthe corpus albicans. This process of regression occupies about four corpus albicans may remain a long time, but the hyalin material is event-ually entirely absorbed and all trace of the former corpus luteum disappears,excepting the scarred indentation on the surface of the ovary. The size of the corpus luteum varies considerably, reaching sometimes even 40 GYNECOLOGY in non-pregnant women a diameter of 2 cm. If pregnancy occurs its develop-ment is more pronounced, reaching its height about the second month. Fromthe third month its regression is very slow, so that it is sometimes demon-strable at the end of pregnancy. If pregnancy is not present, the corpus luteum regresses rapidly and isusually shrunken by the end of a month. Follicle Atresia.—In the thirty to thirty-five years of menstrual life aboutfourteen to eighteen follicles reach full maturity each year, making a total of. Fig. 17.—Graafian Follicle, Well Advanced. 400 to 600 during a life time. Inasmuch as there is an original endowment ofabout 30,000 follicles at birth, only a small percentage of them develop com-pletely, the remainder becoming aborted during some stage of their develop-ment, a process that is called atresia of the follicle. This process must be re-garded as a physiologic one, beginning as it does in the newborn and probablyin the mature fetus and lasting until the menopause. The cause of atresia is thought to be some insufficiency of the local blood- PHYSIOLOGY OF THE UTERUS AND OVARIES 41 supply, or a too deep embedding, which may be unfavorable for the bursting ofthe follicle. Follicle atresia takes place in the following way: The egg slowly undergoes a fatty degeneration and becomes granulosa cells also degenerate by the process of vacuolization and fall intothe central cavity of the follicle. The entire follicle collapses and assumes anirregular conto
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