Eye injuries and their treatment . SERPIGINOUS ULCER OF THE CORNEA 35 to that induced by the diffusion of a chemical poison,and, as a result, the circumcorneal vessels becomedilated, and there is a rapid migration of white bloodcorpuscles through their walls. These leucocytesquickly force their way through the cornea to reachthe spot where the microbes are most active. Thenearer they approach this, however, the more intensebecomes the action of the toxins, and at length, over-powered by the poison, many perish and form a pur-ulent zone round the ulcer, while those which surviveenter the microb
Eye injuries and their treatment . SERPIGINOUS ULCER OF THE CORNEA 35 to that induced by the diffusion of a chemical poison,and, as a result, the circumcorneal vessels becomedilated, and there is a rapid migration of white bloodcorpuscles through their walls. These leucocytesquickly force their way through the cornea to reachthe spot where the microbes are most active. Thenearer they approach this, however, the more intensebecomes the action of the toxins, and at length, over-powered by the poison, many perish and form a pur-ulent zone round the ulcer, while those which surviveenter the microbe area to wage war against the to this view the cornea is perfectly passive,and the leucocytes wander through it at will, to over-come the action of the microbes, or to be themselvesovercome and to meet their death by poisoning in theneighbourhood of the infected area. The cornea is not, however, a passive agent; on thecontrary, it is intensely active, and its protoplasmicelements respond at once to the very slight
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Keywords: ., bo, bookcentury1900, bookdecade1900, bookideyeinjuriestheir00rams
