Modern diagnosis and treatment of diseases of childern; a treatise on the medical and surgical diseases of infancy anf childhood . -spinal .Meningitis/ page 335 I. ( Occasionally, however, the inflam-mation is limited to the spinal membranes, like spinal hemorrhage,being produced by traumatism. The symptoms of spinal meningitis are practically the same asrigidity and \u Spinal hemorrhage, except that the former affection is marked paralysis. « o • r by a sharp rise in temperature at the onset, and by a moreprogressive character of the symptoms. Recovery is treatment is symptoma
Modern diagnosis and treatment of diseases of childern; a treatise on the medical and surgical diseases of infancy anf childhood . -spinal .Meningitis/ page 335 I. ( Occasionally, however, the inflam-mation is limited to the spinal membranes, like spinal hemorrhage,being produced by traumatism. The symptoms of spinal meningitis are practically the same asrigidity and \u Spinal hemorrhage, except that the former affection is marked paralysis. « o • r by a sharp rise in temperature at the onset, and by a moreprogressive character of the symptoms. Recovery is treatment is symptomatic. Pain, fever, SPINAL PARALYSIS. 529 SPINAL PARALYSIS (Poliomyelitis Anterior, Infantile Paralysis). As the name indicates the pathologic anatomy of this affectionconsists of multiple inflammatory foci (hyperemia, edema, infil-tration of the small cells, swelling and cloudiness of the ganglioncells, destruction of the nerve elements, etc.), principally in thegray substance of the anterior horns of the spinal cord. Occa-sionally the inflammation extends to the anterolateral tracts and Lesionsprincipallyin Fig. 168.—Anterior Poliomyelitis, Involving Right atrophy. (Sheffield.) posterior horns, and while, as a rule, the lesion is limited to thecervical or lumbar enlargement or both, it may be found also inother regions of the cord and even in the medulla and pons—hencethe diversity of the symptomatology. After abatement of the acute inflammatory process, some of ^J^the affected portions of the cord usually (there arc but few ,;[.,;,!;exceptions) remain more or less permanently injured (atro-phied ), and it is upon the extent of this permanent—and not uponthe initial—lesion that the further course of the disease depends. It is now generally agreed that the disease, whether it occurs-sporadically or in epidemic form, i^ the result of invasion of the 34 ,30 DISEASES OE THE XERVE SYSTEM. Microbicorigin. spinal cord by a micro-organism or its toxi
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