The Journal of laboratory and clinical medicine . ondition anddied within three weeks. At autopsy extensive atheromatosis with ulceratinj,plaques was found in the aorta, together with carcinomatous nodules in thiprostatic bed, malignant proliferation of the epithelium and the sebaceous glandsin the suprapubic scar. The skin was not broken! It will be remembered thatartificial atheromatosis has been produced in animals by prolonged cholesterinfeeding (Anitschkow, Saltykow, Stuckey, McMeans) and that the atheromatousplaques were microscopically identical with those found in man. Proceeding on th
The Journal of laboratory and clinical medicine . ondition anddied within three weeks. At autopsy extensive atheromatosis with ulceratinj,plaques was found in the aorta, together with carcinomatous nodules in thiprostatic bed, malignant proliferation of the epithelium and the sebaceous glandsin the suprapubic scar. The skin was not broken! It will be remembered thatartificial atheromatosis has been produced in animals by prolonged cholesterinfeeding (Anitschkow, Saltykow, Stuckey, McMeans) and that the atheromatousplaques were microscopically identical with those found in man. Proceeding on the working hypothesis that essential factors for the effectualprogress of malignant proliferation are, (1) the creation of a disturbance ofmetabolism which would lead to an accumulation of cholesterin, (2) an extrane-ous irritant, and (3) the breaking down of the lymphoid-defence, the impor-tance of which has been established by the work of Murphy and Morton,** I be-gan in March, 1915, to give light treatment by roentgen rays to a double-adrenal-.
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