Lectures on nervous diseases from the standpoint of cerebral and spinal localization, and the later methods employed in the diagnosis and treatment of these affections . - of the auricle orventricle may be tleveloped to com[>ensate for the deficiency. In thiscase the organ exhibits no marked impairment of its function; but,when the compensation is disproportionate to the valvular lesion, thereturn of blood from the head is seriously interfered with. Under suchcircumstances anything that tends to over-excite or weaken the heartsaction produces marked cerebral disturbances. 5. S^mptoms of cer
Lectures on nervous diseases from the standpoint of cerebral and spinal localization, and the later methods employed in the diagnosis and treatment of these affections . - of the auricle orventricle may be tleveloped to com[>ensate for the deficiency. In thiscase the organ exhibits no marked impairment of its function; but,when the compensation is disproportionate to the valvular lesion, thereturn of blood from the head is seriously interfered with. Under suchcircumstances anything that tends to over-excite or weaken the heartsaction produces marked cerebral disturbances. 5. S^mptoms of cerebral congestion are occasionally developed as. Fig. 78.—Diagram of a Tkansversb Vertical Section of the Left Cerebral Hemi-sphere, SHOWING THE ARTERIAL DISTRIBUTION. (Modified slightly from We-stbrook.)1, arteries ramifying in the pia, and sending off cortical and medullary branches ; 2, graymatter of ; 3, corpus callosum; 4, cavity of the ventricle; 5, caudate nucleus;6, 6, 6, members of lenticular nucleus (Glieder) ; 7, internal capsule ; 8, septum ; 9, opticchiasm ; 10, arteries from circle of Willis, sending branches to basal ganglia ; 11, convolu-tions of Island of Reil; 12, claustrum. the result of plethora, caused by excess in eating or drinking. It isquestionable to in^^ mind if this form is not always associated with someorganic changes in the arteries,—probably of the atheromatous type. 6. Persistent Anxiety or Emotional Excitement.—This is perhaps themost common and potent of all the etiological fiictors of cerebral conges-tion. The prolonged activity of the brain protracts the determinationof blood to the
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Keywords: ., bookcentury1800, bookdecade1880, bookidlecturesonne, bookyear1888
