A manual of diseases of the nervous system . places where the axis-cylinder is swollen, thisappears much larger than normal (Fig. 93, V). Examples of thiswill be found in most of the figures. These intense degenerativechanges cause the breaking up of the myelin, and may involve thedestruction of the fibre, the space occupied by it being left empty. * The significance of this vacuolation has been much discnssed. It is probablethat tlie vacuoles form after death, perhaps during the process of hardening, butthat ihey do not form unless the cells have been altered by disease. They have thussome pa
A manual of diseases of the nervous system . places where the axis-cylinder is swollen, thisappears much larger than normal (Fig. 93, V). Examples of thiswill be found in most of the figures. These intense degenerativechanges cause the breaking up of the myelin, and may involve thedestruction of the fibre, the space occupied by it being left empty. * The significance of this vacuolation has been much discnssed. It is probablethat tlie vacuoles form after death, perhaps during the process of hardening, butthat ihey do not form unless the cells have been altered by disease. They have thussome pathological bigniticauce. 308 SPINAL CORD. In other cases, however, the axis-cylinder persists and. remains sur-rounded by a narrow zone of myelin. Often a large number of suchshrunken fibres may be found in a part which at first sight appeareddevoid of fibres. This shrinking of the fibres occurs not only as theresult of pressure on them by the connective-tissue elements, but alsowhere there is little interstitial change and no
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Keywords: ., bookcentury1800, bookdecade1890, booksubjectnervoussystem, bookye