Diseases of cattle, sheep, goats Diseases of cattle, sheep, goats and swine diseasesofcattle00mous Year: 1920 650 RINGWORM. neigbourhood of the lips, the nostrils and sub-maxillai7 region, as well as the throat and neck. It assumes the form of circular patches, over which the hair stands erect. Gruby in 1842 discovered the parasite of tinea tonsurans, or herpes, and thus proved that the cutaneous lesions were not due to any constitutional condition, as was long thought, although dirt, bad hygienic conditions, and crowded stables favoured the spread of ring- worm. Direct contact between healt


Diseases of cattle, sheep, goats Diseases of cattle, sheep, goats and swine diseasesofcattle00mous Year: 1920 650 RINGWORM. neigbourhood of the lips, the nostrils and sub-maxillai7 region, as well as the throat and neck. It assumes the form of circular patches, over which the hair stands erect. Gruby in 1842 discovered the parasite of tinea tonsurans, or herpes, and thus proved that the cutaneous lesions were not due to any constitutional condition, as was long thought, although dirt, bad hygienic conditions, and crowded stables favoured the spread of ring- worm. Direct contact between healthy and diseased animals and the transport of spores, by combs, brushes, etc., favour contagion. The disease may not only be conveyed from one animal to another of the same species, but from the ox to man, and, with somewhat greater difficulty, from the ox to the horse. Cases of transmission from the ox to the sheep, pig, and dog have also been recorded. Megnin in 1890 attempted to prove that all the trichophytons pro- ducing ringworm in animals do not belong to the same species, and gave the name of Tricliophyton epilans to that usually found in the ox, be- cause it causes absolute loss of the hair by growing in the follicle, whilst he named the parasite found in the horse Trichophyton tonsurans, be- cause it only grows on the surface of the skin and in the thickness of the hair, without causing inflammation of the hair follicle and without invading it. The epidermis soon undergoes proliferation, and becomes covered with crusts, which adhere to the hairs, gluing them together, and finally causing them to be shed, leaving bare patches the size of a shilling or a florin. The lesion extends in an ever-widening circle, until it attains, perhaps, the dimensions of a tive-shilling piece or more. The affected hairs break off level with the free surface of the skin, rendering the patches more apparent. White hairs are less affected, and some always remain projecting above the crusts, c


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